崔欢欢Google Scholar ResearcherID

研究助理教授 前沿与交叉科学研究院
崔欢欢博士于2016年获得柏林自由大学博士学位，2016-2017年在柏林夏洛蒂医科大学实验与临床研究中心做博士后研究，2017-2018年在华大基因任遗传咨询师和讲师，2018年底加入南方科技大学前沿与交叉科学研究院。
个人简介
个人简介

研究领域染色体重塑、组蛋白修饰等表观遗传因子和机制在发育（如早期发育，心肌分化）和疾病（如肿瘤）中的作用。

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学术成果 查看更多1. Cui H, Schlesinger J, Schoenhals S,T?njes M, Dunkel I, Meierhofer D, Cano E, Schulz K, Berger MF, Haack T, Abdelilah-Seyfried S, Bulyk ML, Sauer S, Sperling SR. Phosphorylation of the chromatin remodeling factor DPF3a induces cardiac hypertrophy through releasing HEY repressors from DNA. 2016. Nucleic Acids Research.
2. Cui H, Bansal V, Schulz K, Malecova B, Dall’Agnese A, Latella L, Gatto S, Chen W, Grunert M, Dorn C, Puri PL, and Sperling SR. Muscle-relevant genes marked by stable H3K4me2/3 profiles and enriched MyoD binding during myogenic differentiation. 2017. Plos One.
3. Grunert M, Dorn C, Cui H, Dunkel I, Schulz K, Schoenhals S, Sun W, Berger F, Chen W, Sperling SR. DNA methylation alterations associated with gene expression changes depict congenital heart disease: Tetralogy of Fallot and ventricular septal defects. 2016. Cardiovascular Research.
4. Ben Jehuda R, Eisen B, Shemer Y, Mekies L.N, Szantai A, Reiter I, Cui H, Guan K, Haron-Khun S, Freimark D, Sperling S.R, Gherghiceanu M, Arad M, Binah O. CRISPR correction of the PRKAG2 gene mutation in the patient’s iPSC-derived cardiomyocytes eliminates the electrophysiological and structural abnormalities. 2017. Heart Rhythm.



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