个人简介

2000 - 2004 四川大学， 学士
2004 - 2010 上海生命科学研究院，博士
2011 - 2014 美国南加州大学，博士后
2014 - 至今 重庆大学“****”教授，博士生导师




研究方向
代谢与衰老生物学。衰老是生命发展的必经阶段，衰老与许多慢性疾病的产生，如癌症，肥胖和二型糖尿病等，有着密切的关系，这些疾病因此也可以称为衰老相关性疾病。本研究组以模式动物研究为基础，致力于解析衰老的遗传和代谢机制，探索寿命决定的分子基础，并以此为基础探索延缓衰老的途径。

主讲课程
本科生《遗传学》，《免疫学》

主要成果
代表性论文（* correspondence）
1. He B, Xu J, Pang S*, Tang H*. Phosphatidylcholine mediates the crosstalk between LET-607 and DAF-16 stress response pathways. PLoS Genet. 2021, 17(5): e**. (自然指数期刊)
2. Deng J#, Bai X#, Tang H*, Pang S*. DNA damage promotes ER stress resistance through elevation of unsaturated phosphatidylcholine in C. elegans. J Biol Chem. 2021, 296:100095.（自然指数期刊）
3. Deng J, Dai Y, Tang H*, Pang S*. SKN-1 Is a Negative Regulator of DAF-16 and Somatic Stress Resistance in Caenorhabditis elegans. G3 (Bethesda). 10(5):1707-1712. (2020)
4. Zhou L, He B, Deng J, Pang S*, Tang H*. Histone acetylation promotes long-lasting defense responses and longevity following early life heat stress. PLoS Genet. 2019, 15(4): e**. (自然指数期刊)
5. Tang H*, Pang S*. Proline catabolism modulates innate immunity in C. elegans. Cell Reports. 2016, 17(11): 2837-2844. (Cell子刊)
6. Pang S#, Lynn D#, Lo J, Curran SP. SKN-1 and Nrf2 couples proline catabolism with lipid metabolism during nutrient deprivation. Nature Communications. 2014, 5:5048. (Nature子刊)
7. Pang S, Curran SP. Adaptive capacity to bacterial diet modulates aging in C. elegans. Cell Metabolism. 2014, 19(2): 221-231. (Cell子刊). (Previewed as a leading edge finding in Cell Metabolism)
8. Pang S, Tang H, Zhuo S, Zang YQ and Le Y. Regulation of fasting fuel metabolism by Toll-like receptor 4. Diabetes. 2010, 59: 3041-3048.

联系方式
邮箱：sspang@cqu.edu.cn